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A team of researchers from the University of Washington in the USA unveiled that the cystic fibrosis transmembrane conductance regulator (CFTR) protein, serving as a chloride channel activator, confers benefits upon a subset of susceptible long-term care residents grappling with dementia. This revelation underscores the role of the PD-L1 protein, or programmed death ligand-1, and its engagement with its programmed death receptor counterpart, PD-1.
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